Hengheng DAI, Haisong LI, Junlong FENG, et al. Prof. Li Yueqing’s experience with Morinda officinalis*. [J]. Modern Chinese Clinical Medicine 29(1):22-24(2022)
DOI:
Hengheng DAI, Haisong LI, Junlong FENG, et al. Prof. Li Yueqing’s experience with Morinda officinalis*. [J]. Modern Chinese Clinical Medicine 29(1):22-24(2022) DOI: 10.3969/j.issn.2095-6606.2022.01.005.
Prof. Li Yueqing’s experience with Morinda officinalis*
a famous master of traditional Chinese medicine in Beijing
with clinical practice for more than 50 years
has rich clinical experience. Professor Li has unique insights into the use of many medicinals. Take Morinda officinalis as an example. He has made good use of its characteristics of invigorating and soothing to treat male diseases. The combination of Morinda officinalis and Achyranthes bidentata can invigorate the kidney and promote blood circulation and can be used to treat male erectile dysfunction. The compatibility of Morinda officinalis and Cuscuta can invigorate the kidney and promote spermatogenesis
and can be used to treat male infertility. The compatibility of Morinda officinalis and Eucommia ulmoides has the effect of strengthening muscles and bones
and can be used to treat low back pain due to fatigue. The combination of Morinda officinalis and turmeric
with the effect of invigorating and soothing
can be used to treat chronic prostatitis. The compatibility of Morinda officinalis and Polyporus umbellatus
with the effect of replenishing the center
can be used to treat benign prostatic hyperplasia. Professor Li has an orderly heritage and is good at innovations. He is unique in the use of Morinda officinalis.
XU L Z, XU D F, HAN Y, et al. BDNF-GSK-3β-β-Catenin pathway in the mPFC is involved in antidepressant-like effects of morinda officinalis oligosaccharides in rats[J].International Journal of Neuropsychopharmacology,2017,20(1):83-93.
QIU Z K, LIU C H, GAO Z W, et al. The inulin-type oligosaccharides extract from morinda officinalis,a traditional Chinese herb,ameliorated behavioral deficits in an animal model of post-traumatic stress disorder[J]. Metabolic Brain Disease,2016,31(5):1143-1149.